Non-plaque-induced gingival lesions

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Non-plaque-induced gingival lesions. / Holmstrup, P.

In: Subject Guide to Books in Print, Vol. 4, No. 1, 01.12.1999, p. 20-31.

Research output: Contribution to journalJournal articleResearchpeer-review

Harvard

Holmstrup, P 1999, 'Non-plaque-induced gingival lesions', Subject Guide to Books in Print, vol. 4, no. 1, pp. 20-31. https://doi.org/10.1902/annals.1999.4.1.20

APA

Holmstrup, P. (1999). Non-plaque-induced gingival lesions. Subject Guide to Books in Print, 4(1), 20-31. https://doi.org/10.1902/annals.1999.4.1.20

Vancouver

Holmstrup P. Non-plaque-induced gingival lesions. Subject Guide to Books in Print. 1999 Dec 1;4(1):20-31. https://doi.org/10.1902/annals.1999.4.1.20

Author

Holmstrup, P. / Non-plaque-induced gingival lesions. In: Subject Guide to Books in Print. 1999 ; Vol. 4, No. 1. pp. 20-31.

Bibtex

@article{daa562b946e2422ca6f0eccbf378907b,
title = "Non-plaque-induced gingival lesions",
abstract = "The origin of gingival inflammation is occasionally different from that of routine plaque-associated gingivitis, and such non-plaque-associated types of gingivitis often present characteristic clinical features. Examples of such forms of gingivitis are specific bacterial, viral, and fungal infections. Specific bacterial infections of gingiva may be due to Neisseria gonorrhea, Treponema pallidum, streptococci, and other organisms. The most important viral infections of gingiva are herpes simplex virus type 1 and 2 and varicella-zoster virus. Fungal infections may be caused by several fungi, the most important of these being Candida species including C. albicans, C. glabrata, C. krusei, C. tropicalis, C. parapsilosis, and C. guillermondii. Gingival histoplasmosis is a granulomatous disease caused by the fungus Histoplasma capsulatum and, as for the other specific infections of gingiva, a confirmed diagnosis may require histopathologic examination and/or culture. Atypical gingivitis may also occur as gingival manifestations of dermatological diseases, the most relevant of these being lichen planus, pemphigoid, pemphigus vulgaris, erythema multiforme, and lupus erythematosus. Non-plaque induced gingival inflammation can be caused by allergic reactions to dental restorative materials, toothpastes, mouthwashes, and foods. In addition, gingival inflammation may result from toxic reactions, foreign body reactions, or mechanical and thermal trauma.",
keywords = "Communicable Diseases, Dental Plaque, Foreign-Body Reaction, Gingival Diseases, Hot Temperature, Humans, Hypersensitivity, Skin Diseases",
author = "P Holmstrup",
year = "1999",
month = dec,
day = "1",
doi = "10.1902/annals.1999.4.1.20",
language = "English",
volume = "4",
pages = "20--31",
journal = "Subject Guide to Books in Print",
issn = "0000-0159",
publisher = "Grey House Publishing",
number = "1",

}

RIS

TY - JOUR

T1 - Non-plaque-induced gingival lesions

AU - Holmstrup, P

PY - 1999/12/1

Y1 - 1999/12/1

N2 - The origin of gingival inflammation is occasionally different from that of routine plaque-associated gingivitis, and such non-plaque-associated types of gingivitis often present characteristic clinical features. Examples of such forms of gingivitis are specific bacterial, viral, and fungal infections. Specific bacterial infections of gingiva may be due to Neisseria gonorrhea, Treponema pallidum, streptococci, and other organisms. The most important viral infections of gingiva are herpes simplex virus type 1 and 2 and varicella-zoster virus. Fungal infections may be caused by several fungi, the most important of these being Candida species including C. albicans, C. glabrata, C. krusei, C. tropicalis, C. parapsilosis, and C. guillermondii. Gingival histoplasmosis is a granulomatous disease caused by the fungus Histoplasma capsulatum and, as for the other specific infections of gingiva, a confirmed diagnosis may require histopathologic examination and/or culture. Atypical gingivitis may also occur as gingival manifestations of dermatological diseases, the most relevant of these being lichen planus, pemphigoid, pemphigus vulgaris, erythema multiforme, and lupus erythematosus. Non-plaque induced gingival inflammation can be caused by allergic reactions to dental restorative materials, toothpastes, mouthwashes, and foods. In addition, gingival inflammation may result from toxic reactions, foreign body reactions, or mechanical and thermal trauma.

AB - The origin of gingival inflammation is occasionally different from that of routine plaque-associated gingivitis, and such non-plaque-associated types of gingivitis often present characteristic clinical features. Examples of such forms of gingivitis are specific bacterial, viral, and fungal infections. Specific bacterial infections of gingiva may be due to Neisseria gonorrhea, Treponema pallidum, streptococci, and other organisms. The most important viral infections of gingiva are herpes simplex virus type 1 and 2 and varicella-zoster virus. Fungal infections may be caused by several fungi, the most important of these being Candida species including C. albicans, C. glabrata, C. krusei, C. tropicalis, C. parapsilosis, and C. guillermondii. Gingival histoplasmosis is a granulomatous disease caused by the fungus Histoplasma capsulatum and, as for the other specific infections of gingiva, a confirmed diagnosis may require histopathologic examination and/or culture. Atypical gingivitis may also occur as gingival manifestations of dermatological diseases, the most relevant of these being lichen planus, pemphigoid, pemphigus vulgaris, erythema multiforme, and lupus erythematosus. Non-plaque induced gingival inflammation can be caused by allergic reactions to dental restorative materials, toothpastes, mouthwashes, and foods. In addition, gingival inflammation may result from toxic reactions, foreign body reactions, or mechanical and thermal trauma.

KW - Communicable Diseases

KW - Dental Plaque

KW - Foreign-Body Reaction

KW - Gingival Diseases

KW - Hot Temperature

KW - Humans

KW - Hypersensitivity

KW - Skin Diseases

U2 - 10.1902/annals.1999.4.1.20

DO - 10.1902/annals.1999.4.1.20

M3 - Journal article

C2 - 10863372

VL - 4

SP - 20

EP - 31

JO - Subject Guide to Books in Print

JF - Subject Guide to Books in Print

SN - 0000-0159

IS - 1

ER -

ID: 35267418